| The UK Health Education Council tells us that almost 40 per cent of all deaths of people between the ages of 35 and 74 arise as the result of stroke or heart attack. The majority of these crises result from circulatory restrictions or obstructions which are both preventable and treatable by chelation therapy.
Those problems arising from atherosclerosis are far and away the greatest health problem in industrialized societies. And as most people now know, cardiovascular and circulatory degenerative changes are to a very great extent preventable, since it is now well established that life-style practices and dietary habits contribute significantly towards their causation. So not only are most diseases which stem from circulatory degeneration largely preventable, in many instances they are at least partly reversible if causative factors are stopped and positive action taken.
However, the range of contributory factors is very wide indeed and no single method of prevention can possibly cover all of them, ranging as they do from the unavoidable - inherited tendencies, age and sex - to the (usually) controllable - smoking, dietary habits, stress coping abilities and exercise patterns. It has been demonstrated, in the Pritikin programme for example (Pritikin 1980), that much can be achieved through the application of self-applied dietary strategies, avoidance of known irritants (smoke, high-fat diet, etc.), combined with the application of aerobic exercise methods. Just how such methods can help, either on their own, or as part of a wider; therapeutic approach - whether this involves drugs, surgery or chelation therapy - will become clearer once the known causes of circulatory obstruction are examined.
The value of chelation as an intervention strategy will also be seen to have marked advantages over many 'high-tech' approaches, in such conditions, once we comprehend that the entire arterial network is frequently damaged, requiring a method of treatment which addresses all 40,000 miles of it, rather than just local, isolated points of major blockage receiving attention.
The Birth of an Atherosclerotic Lesion
There is no absolute consensus as to the causes of atherosclerosis, which probably means that all, or a number, of the theories are at least partially correct. It is therefore necessary to examine the most popular of these hypotheses.
In good health an artery (or arteriole) is far more than a simple plumbing conduit. As with so many parts of the body it also acts as a mini-factory, producing a large number of vital biochemical agents such as enzymes which act to protect it from damage which could arise via the action of a number of agencies (see below), such as excess fat in the bloodstream or other potential sources of free radical activity (see Chapter 2). The ability of such enzymes to perform their defensive and other functions depends on the abundant presence of co-factors vitamins such as A, C, E, D) and an army of minerals and adequate protein sources for the amino and nucleic acids needed for regeneration and repair functions.
Vulnerability/susceptibility
Nutritional excellence is therefore the essential background to all other potential causes of arterial damage. If the nutritional status of the region is sound, the resulting abundant supply of defending substances will provide a powerful protective shield. Conversely, if nutrition is poor, vulnerability is greater and far fewer and lesser stress factors will be required before serious damage is caused.
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